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Specimen Atlas of Research Peptides81 plates · MIT
Side-by-side · Research reference

FOXO4-DRIvsHGH Fragment 176-191

Side-by-side comparison across mechanism, dosage, evidence, side effects, administration, and stack synergies. Citations on every claim where available.

AAnimal-StrongHUMAN-REVIEWED12/45 cited
BAnimal-StrongHUMAN-REVIEWED28/59 cited
FOXO4-DRI
Senolytic Peptide · D-Retro-Inverso
p53-TADMolecular targetBourgeois 2025
Pre-clinicalDevelopment stage
SQRoute (animal)
SQ · Animal models only
HGH Fragment 176-191
GH Fragment · Pre-Clinical
50%Weight gain reductionNg 2000
~26 minHalf-life (est.)
No IGF-1 ↑GH axis impact
SQ · IP (animal) · Oral (tested)

01Mechanism of Action

Parameter
FOXO4-DRI
HGH Fragment 176-191
Primary target
FOXO4-p53 protein complex in senescent cellsBourgeois 2025Tripathi 2021
Beta-3 adrenergic receptors on adipocytesHeffernan 2001
Pathway
FOXO4-DRI binds disordered p53 transactivation domain → displaces FOXO4 → nuclear p53 exclusion → p53-mediated apoptosis in senescent cells
Fragment → β3-AR upregulation → Enhanced lipolytic sensitivityHeffernan 2001
Downstream effect
Selective apoptosis of senescent cells; clearance restores tissue homeostasisTripathi 2021Alameen 2026
Increased lipolysis and beta-3 AR mRNA expression without IGF-1 axis activation
Feedback intact?
N/A — does not interact with GH/IGF-1 axis
Origin
D-retro-inverso modification — inverted amino acid sequence, D-amino acids for protease resistance
Synthetic peptide derived from hGH residues 176-191; AOD9604 includes N-terminal tyrosine (177-191)Cox 2015
Antibody development
Not reported in available studies

02Dosage Protocols

Parameter
FOXO4-DRI
HGH Fragment 176-191
Animal dose (mouse)
5 mg/kg
SQ injection, aged mouse model (testosterone restoration).
Frequency (animal)
Variable — single or intermittent dosing
Protocol-dependent; no standardised regimen.
Human equivalent (theoretical)
~0.4 mg/kg (28 mg / 70 kg adult)
Extrapolated using allometric scaling; no clinical validation.
Evidence basis
Animal / mechanistic
Animal studies only
Route
SQ (animal)
No human route established.
Duration
Weeks to months (animal studies)
Senescent cell clearance observed within weeks.
Clinical status
No human trials completed
Animal dose (oral)
500 mcg/kg body weightNg 2000
Obese Zucker rats, 19 days.
Animal dose (IP)
Not specified (14-day chronic administration)Heffernan 2001
Obese mice, daily IP injection.
Human equivalent dose
Not established — no published human RCTs
Frequency
Once daily (animal models)
Duration tested
Detection window
50 pg/mL LOD in urine; stable metabolite extends detectionCox 2015
WADA-banned; anti-doping testing available.
Oral bioavailability
Demonstrated efficacy in animal oral administrationNg 2000
Potential for oral therapeutic development.

03Metabolic / Fat Loss Evidence

Parameter
FOXO4-DRI
HGH Fragment 176-191
Primary fat target
Adipose tissue (general) — beta-3 AR mediated lipolysisHeffernan 2001
Weight gain reduction
50% reduction vs control (15.8 ± 0.6 g vs 35.6 ± 0.8 g)Ng 2000
Obese Zucker rats, 19 days oral administration.
Body fat reduction
Significant decrease in body weight and body fat in obese mice (14 days)Heffernan 2001
Lipolytic activity
Increased adipose tissue lipolytic activityNg 2000
Direct measurement in treated animals.
Beta-3 AR expression
Upregulated β3-AR mRNA in obese mice to lean-comparable levelsHeffernan 2001
Insulin sensitivity
No adverse effect — euglycemic clamp confirmedNg 2000
Contrasts with intact hGH diabetogenic effects.
IGF-1 impact
No elevation — fragment does not activate GH/IGF-1 axis
Beta-3 AR dependency
Effect abolished in β3-AR knockout miceHeffernan 2001
Confirms β3-AR as primary mechanism.
Route of administration
Efficacy demonstrated via oral and IP routesNg 2000Heffernan 2001
Human evidence
None published — pre-clinical only

04Side Effects & Safety

Parameter
FOXO4-DRI
HGH Fragment 176-191
Pulmonary hypertension risk
Senescent cell elimination promoted PH development/progression in rodent modelsBorn 2023
Context-dependent toxicity
Beneficial effects may be tissue/context-specific; elimination not universally protectiveBorn 2023
Off-target apoptosis
Theoretical risk of non-senescent cell apoptosis (selectivity not absolute)
Immune perturbation
Senescent cells contribute to immune surveillance; clearance effects unknown
Human safety unknown
No clinical trials — toxicity profile in humans not established
Insulin sensitivity
No adverse effects observed in euglycemic clamp (animal)Ng 2000
GH/IGF-1 axis
No activation — avoids diabetogenic effects of full GHNg 2000
Human safety data
Not available — no published human trials
WADA status
Banned as performance-enhancing drugCox 2015
Metabolic profile
Six metabolites identified; CRSVEGSCG most stableCox 2015
Detection window implications for doping control.
Absolute Contraindications
FOXO4-DRI
  • ·Pulmonary hypertension or vascular disease (preclinical evidence of harm)Born 2023
  • ·Pregnancy / lactation (no safety data)
HGH Fragment 176-191
  • ·Competitive athletes (WADA-banned)Cox 2015
Relative Contraindications
FOXO4-DRI
  • ·Active malignancy (senescence as tumour suppressor mechanism)
  • ·Wound healing / tissue repair (senescent cells involved in fibrosis resolution)
HGH Fragment 176-191
  • ·Absence of human safety data — experimental use only

05Administration Protocol

Parameter
FOXO4-DRI
HGH Fragment 176-191
1. Pre-clinical route
Subcutaneous injection used in rodent models. No human administration protocol exists.
Subcutaneous injection primary route in research context. Oral administration demonstrated efficacy in animal models at 500 mcg/kg.
2. Reconstitution (animal)
Typically reconstituted in sterile saline or PBS for animal experiments. Stability data limited.
Once daily dosing used in animal studies. Timing not specified; GH-independent mechanism suggests flexibility.
3. Dosing schedule
Variable — single bolus or intermittent dosing over weeks. No standardised human protocol.
Animal protocols: 14–19 days. Human duration not established — no published trials.
4. Clinical development status
No registered human trials. Commercialisation by Cleara Biotech (Netherlands) in development phase.
Lyophilized peptide storage per standard peptide protocols. Metabolite stability suggests refrigerated reconstituted solution viable.
5. Safety monitoring (proposed)
Would require cardiovascular assessment, pulmonary function, immune panel, tumour surveillance if human trials proceed.
Detectable in urine via SPE-LC-MS at 50 pg/mL LOD. Extended detection window via stable metabolite CRSVEGSCG.Cox 2015