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Specimen Atlas of Research Peptides81 plates · MIT
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XXVIIIPlate XXVIIIReviewed 2026-04-27

GLP-1 (7-37)

Incretin Hormone

also known as Glucagon-like peptide-1, GLP-1(7-37), Native GLP-1

Native 31-amino-acid incretin hormone secreted by intestinal L cells in response to nutrient ingestion. Stimulates glucose-dependent insulin secretion, suppresses glucagon, and delays gastric emptying. Rapidly degraded by dipeptidyl peptidase-4 (DPP-4) with plasma half-life of ~2 minutes. Parent molecule from which all clinical GLP-1 receptor agonists (semaglutide, liraglutide, dulaglutide, exenatide) were engineered. Direct administration rarely used clinically; studied primarily as reference standard for analogue development.

§ I

At a glance

Molecular weight
3297.7 Da
Discovery year
1922
Route

Research use only · IV/SC in experimental settings

§ II

Mechanism

Edit ↗

Primary target — GLP-1 receptor (class B GPCR) [koole-2015].

Pathway — GLP-1R activation → cAMP production → PKA signaling → insulin secretion (pancreatic β-cells) [lu-2025][koole-2015].

Downstream effect — Glucose-dependent insulin release, glucagon suppression, delayed gastric emptying, reduced food intake [lu-2025][ding-2017].

Origin — Endogenous peptide cleaved from proglucagon in intestinal L cells; secreted postprandially.

Feedback intact — Yes — physiological secretion and degradation preserved.

§ III

Dosage

Protocols described in the cited literature; not medical advice.

Edit ↗
ParameterValue
Clinical useNone — native GLP-1 not used therapeuticallyEngineered analogues (semaglutide, liraglutide) used clinically. [friedman-2024]
Research dosingVariable — 0.1–10 nmol/kg in animal modelsUsed as reference standard for analogue comparison.
Half-life~2 minutes (plasma) [alavi-2021][ding-2017]Requires continuous infusion for sustained effect.
Modified analoguest½ extended to 13 h (liraglutide), 165 h (semaglutide)Via DPP-4 resistance + fatty acid acylation.
§ III · b

Reconstitution

A pure mass-to-volume utility. Enter what you have in the vial; the atlas computes the volume per dose. No prescription information.

Inputs
mg
mL
mcg
The calculator does pure mass-to-volume math. It does not recommend a dose. Refer to GLP-1 (7-37)'s cited literature for protocol specifics.
Volumetric outputFig. C — reconstitution math
Volume per dose
0.100mL
10.0 units on a U-100 insulin syringe
Concentration
2500
mcg per mL
Doses per vial
20
at this dose
§ IV

Evidence

Edit ↗
Strength
30/100
animal mechanistic

Native GLP-1 not used for fat loss — short half-life precludes sustained effect. Engineered analogues (semaglutide, liraglutide) demonstrate robust fat loss via prolonged GLP-1R activation.

OutcomeFinding
MechanismGLP-1R activation in hypothalamic satiety centers (arcuate nucleus) reduces food intake [lu-2025]Effect demonstrated with long-acting analogues (liraglutide).
Native GLP-1 efficacyMinimal — rapid degradation prevents sustained appetite suppression
Gastric emptyingDelayed in animal models, contributing to satiety
Body weight impactNot observed with native GLP-1 — requires analogue formulations
§ V

Adverse events

Severities follow the FDA / CTCAE convention.

Edit ↗
Native GLP-1mild
Well-tolerated in research settings; no prolonged exposure data
Hypoglycemia riskmild
Low — insulin secretion is glucose-dependent
Analogue side effectsmoderate
Nausea, vomiting, diarrhea (GLP-1R agonists)
GLP-1 resistancemoderate
High glucose-induced PKCβ overexpression may reduce GLP-1 responsiveness in endothelial cells [pujadas-2016]
§ VI

Administration

Edit ↗
  1. 01
    Research use only

    Native GLP-1(7-37) is not formulated for therapeutic use. Administered IV or SC in experimental protocols to study GLP-1R pharmacology and as reference standard for analogue development.

  2. 02
    Storage

    Lyophilised peptide stored at -20°C or below. Reconstituted solutions should be prepared fresh and used immediately due to rapid degradation.

  3. 03
    Clinical alternatives

    For therapeutic GLP-1R activation, use FDA-approved long-acting analogues: semaglutide (once weekly), liraglutide (once daily), dulaglutide (once weekly), or exenatide (twice daily or once weekly).

Appendix

Sources

37%

of 43 rendered claims carry a resolvable citation.

  1. [alavi-2021]
    Alavi 2021Developing GLP-1 Conjugated Self-Assembling Nanofibers Using Copper-Catalyzed Alkyne-Azide Cycloaddition and Evaluation of Their Biological Activity.
    journal, 2021
  2. [cai-2018]
    Cai 2018Novel fatty acid chain modified GLP-1 derivatives with prolonged in vivo glucose-lowering ability and balanced glucoregulatory activity.
    journal, 2018
  3. [ding-2017]
    Ding 2017BPI-3016, a novel long-acting hGLP-1 analogue for the treatment of Type 2 diabetes mellitus.
    journal, 2017
  4. [friedman-2024]
    Friedman 2024The discovery and development of GLP-1 based drugs that have revolutionized the treatment of obesity.
    journal, 2024
  5. [koole-2015]
    Koole 2015Differential impact of amino acid substitutions on critical residues of the human glucagon-like peptide-1 receptor involved in peptide activity and small-molecule allostery.
    journal, 2015
  6. [lu-2025]
    Lu 2025Effects of tryptophan-selective lipidated glucagon-like peptide 1 (GLP-1) peptides on the GLP-1 receptor.
    journal, 2025
  7. [pujadas-2016]
    Pujadas 2016The pivotal role of high glucose-induced overexpression of PKCβ in the appearance of glucagon-like peptide-1 resistance in endothelial cells.
    journal, 2016
  8. [skurikhin-2017]
    Skurikhin 2017Effects of Pegylated Glucagon-Like Peptide-1 Analogue in C57Bl/6 Mice under Optimal Conditions and During Streptozotocin-Induced Diabetes.
    journal, 2017
  9. [wang-2025]
    Wang 2025Design, synthesis, and biological evaluation of long-acting glucagon-like peptide-1 (GLP-1) conjugates modified with dual fatty acids and a proline-alanine-serine (PAS) polypeptide.
    journal, 2025
Plate composed 2026-04-27 · maturity human-reviewed · schema v1 · Contributors: peptidesdb-core · 27 fields uncited — open contributions