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Specimen Atlas of Research Peptides81 plates · MIT
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34Plate 34Reviewed 2026-04-27

HGH Fragment 176-191

GH Fragment

also known as AOD9604, AOD-9604, hGH 176-191

C-terminal fragment of human growth hormone spanning amino acids 176-191, with an additional N-terminal tyrosine residue in the AOD9604 variant. Retains lipolytic activity of parent GH without IGF-1 elevation or diabetogenic effects. Animal studies demonstrate 50% reduction in weight gain and enhanced beta-3 adrenergic receptor expression in obese mice. No adverse effects on insulin sensitivity observed in euglycemic clamp studies.

§ I

At a glance

Weight gain reduction
50%
Half-life (est.)
~26 min
GH axis impact
No IGF-1 ↑
Route

SQ · IP (animal) · Oral (tested)

§ II

Mechanism

Edit ↗

Primary target — Beta-3 adrenergic receptors on adipocytes [heffernan-2001].

Pathway — Fragment → β3-AR upregulation → Enhanced lipolytic sensitivity [heffernan-2001].

Downstream effect — Increased lipolysis and beta-3 AR mRNA expression without IGF-1 axis activation.

Origin — Synthetic peptide derived from hGH residues 176-191; AOD9604 includes N-terminal tyrosine (177-191) [cox-2015].

Feedback intact — N/A — does not interact with GH/IGF-1 axis.

§ III

Dosage

Protocols described in the cited literature; not medical advice.

Edit ↗
ParameterValue
Animal dose (oral)500 mcg/kg body weight [ng-2000]Obese Zucker rats, 19 days.
Animal dose (IP)Not specified (14-day chronic administration) [heffernan-2001]Obese mice, daily IP injection.
Human equivalent doseNot established — no published human RCTs
FrequencyOnce daily (animal models)
Evidence basisAnimal studies only
Duration tested14–19 days [heffernan-2001][ng-2000]
Detection window50 pg/mL LOD in urine; stable metabolite extends detection [cox-2015]WADA-banned; anti-doping testing available.
Oral bioavailabilityDemonstrated efficacy in animal oral administration [ng-2000]Potential for oral therapeutic development.
§ III · b

Reconstitution

A pure mass-to-volume utility. Enter what you have in the vial; the atlas computes the volume per dose. No prescription information.

Inputs
mg
mL
mcg
The calculator does pure mass-to-volume math. It does not recommend a dose. Refer to HGH Fragment 176-191's cited literature for protocol specifics.
Volumetric outputFig. C — reconstitution math
Volume per dose
0.100mL
10.0 units on a U-100 insulin syringe
Concentration
2500
mcg per mL
Doses per vial
20
at this dose
§ IV

Evidence

Edit ↗
Strength
45/100
animal strong

Multiple animal models (obese Zucker rats, obese mice, β3-AR knockout mice) · 14–19 days chronic administration · No human RCTs published

OutcomeFinding
Primary fat targetAdipose tissue (general) — beta-3 AR mediated lipolysis [heffernan-2001]
Weight gain reduction50% reduction vs control (15.8 ± 0.6 g vs 35.6 ± 0.8 g) [ng-2000]Obese Zucker rats, 19 days oral administration.
Body fat reductionSignificant decrease in body weight and body fat in obese mice (14 days) [heffernan-2001]
Lipolytic activityIncreased adipose tissue lipolytic activity [ng-2000]Direct measurement in treated animals.
Beta-3 AR expressionUpregulated β3-AR mRNA in obese mice to lean-comparable levels [heffernan-2001]
Insulin sensitivityNo adverse effect — euglycemic clamp confirmed [ng-2000]Contrasts with intact hGH diabetogenic effects.
IGF-1 impactNo elevation — fragment does not activate GH/IGF-1 axis
Beta-3 AR dependencyEffect abolished in β3-AR knockout mice [heffernan-2001]Confirms β3-AR as primary mechanism.
Route of administrationEfficacy demonstrated via oral and IP routes [ng-2000][heffernan-2001]
Human evidenceNone published — pre-clinical only
§ V

Adverse events

Severities follow the FDA / CTCAE convention.

Edit ↗
Insulin sensitivitymild
No adverse effects observed in euglycemic clamp (animal) [ng-2000]
GH/IGF-1 axis
No activation — avoids diabetogenic effects of full GH [ng-2000]
Human safety datamoderate
Not available — no published human trials
WADA status
Banned as performance-enhancing drug [cox-2015]
Metabolic profile
Six metabolites identified; CRSVEGSCG most stable [cox-2015]
Absolute contraindications
Relative contraindications
  • Absence of human safety data — experimental use only
§ VI

Administration

Edit ↗
  1. 01
    Route

    Subcutaneous injection primary route in research context. Oral administration demonstrated efficacy in animal models at 500 mcg/kg.

  2. 02
    Frequency

    Once daily dosing used in animal studies. Timing not specified; GH-independent mechanism suggests flexibility.

  3. 03
    Duration

    Animal protocols: 14–19 days. Human duration not established — no published trials.

  4. 04
    Storage

    Lyophilized peptide storage per standard peptide protocols. Metabolite stability suggests refrigerated reconstituted solution viable.

  5. 05
    Detection

    Detectable in urine via SPE-LC-MS at 50 pg/mL LOD. Extended detection window via stable metabolite CRSVEGSCG. [cox-2015]

Appendix

Sources

47%

of 59 rendered claims carry a resolvable citation.

  1. [cox-2015]
    Cox 2015Detection and in vitro metabolism of AOD9604.
    journal, 2015
  2. [heffernan-2001]
    Heffernan 2001The effects of human GH and its lipolytic fragment (AOD9604) on lipid metabolism following chronic treatment in obese mice and beta(3)-AR knock-out mice
    Endocrinology, 2001
  3. [ng-2000]
    Ng 2000Metabolic studies of a synthetic lipolytic domain (AOD9604) of human growth hormone.
    journal, 2000
Plate composed 2026-04-27 · maturity human-reviewed · schema v1 · Contributors: peptidesdb-core · 31 fields uncited — open contributions